Search results for "Estradiol Congeners"

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Estrogens increase transcription of the human endothelial NO synthase gene: analysis of the transcription factors involved.

1998

Abstract —Estrogens have been found to reduce the incidence of cardiovascular disease that has been ascribed in part to an increased expression and/or activity of the vasoprotective endothelial NO synthase (NOS III). Some reports have shown that the level of expression of this constitutive enzyme can be upregulated by estrogens. The current study investigates the molecular mechanism of the NOS III upregulation in human endothelial EA.hy 926 cells. Incubation of EA.hy 926 cells with 17β-estradiol or the more stable 17α-ethinyl estradiol enhanced NOS III mRNA and protein expression up to 1.8-fold, without changing the stability of the NOS III mRNA. There was no enhancement of NOS III mRNA af…

Transcription Geneticmedicine.drug_classBiologyEthinyl EstradiolTransfectionCell LineDownregulation and upregulationDrug StabilityEstradiol CongenersTranscription (biology)Consensus SequenceInternal MedicinemedicineHumansRNA MessengerPromoter Regions GeneticTranscription factorCell NucleusSp1 transcription factorMessenger RNABase SequenceEstradiolTissue ExtractsTransfectionDNAMolecular biologyEndothelial stem cellIsoenzymesEstrogenEndothelium VascularNitric Oxide Synthasehormones hormone substitutes and hormone antagonistsTranscription FactorsHypertension (Dallas, Tex. : 1979)
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Neuroprotective Actions of Estradiol and Novel Estrogen Analogs in Ischemia: Translational Implications

2010

This review highlights our investigations into the neuroprotective efficacy of estradiol and other estrogenic agents in a clinically relevant animal model of transient global ischemia, which causes selective, delayed death of hippocampal CA1 neurons and associated cognitive deficits. We find that estradiol rescues a significant number of CA1 pyramidal neurons that would otherwise die in response to global ischemia, and this is true when hormone is provided as a long-term pretreatment at physiological doses or as an acute treatment at the time of reperfusion. In addition to enhancing neuronal survival, both forms of estradiol treatment induce measurable cognitive benefit in young animals. Mo…

medicine.medical_specialtyGPR30hippocampusIschemiaEstrogen receptorHippocampusNeuroprotectionArticleEstradiol CongenersIschemiaInternal medicineestradiolmedicineAnimalsHumansEndocrine and Autonomic Systemsbusiness.industryCREBEstrogen analogapoptosismedicine.diseasestrokeglobal ischemiainsulin-like growth factor-1EndocrinologyNeuroprotective AgentsneuroprotectionEstradiol CongenersbusinessGPERhormones hormone substitutes and hormone antagonistsHormoneestrogen receptor
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